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The inhibition of PDE5 in A few of these tissues by sildenafil may be the basis for the enhanced platelet antiaggregatory exercise of NO observed in vitro, an inhibition of platelet thrombus development in vivo and peripheral arterial-venous dilatation in vivo.
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In balanced male volunteers, there was no evidence of the clinically substantial influence of azithromycin (five hundred mg everyday for three days) over the systemic exposure of sildenafil or its big circulating metabolite.
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Just after possibly oral or intravenous administration, sildenafil is excreted as metabolites predominantly within the feces (around 80% of administered oral dose) and to a lesser extent in the urine (approximately thirteen% of your administered oral dose).
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